Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. Then again, our ability to rescue SERT purpose in KI midbrain synaptoneurosomes by the inhibition of FAK suggests elevated FAK signaling downstream from the Pro32Pro33 mutant, as confirmed by greater pFAK localization in 5-HT synapses. Our https://rudyardm541pcm3.vidublog.com/profile